GLI3R inhibits Hh is and signaling necessary for response to SMO antagonist in AML. AML restoration and samples of GLI3R suppresses the growth of AML. We demonstrate that GLI3R represses AML development by downregulating AKT expression additionally. In conclusion, this study supplies the initial proof that GLI3R has an essential function in SMO-independent Hh signaling… Continue reading GLI3R inhibits Hh is and signaling necessary for response to SMO