Alzheimer’s disease (AD) is pathologically seen as a accumulation of β-amyloid (Aβ) protein Rabbit Polyclonal to NMBR. deposits and/or neurofibrillary tangles in association with progressive cognitive deficits. Aβ vaccinations reduced retinal Aβ deposits there was a marked increase in retinal microvascular Aβ deposition as well as local neuroinflammation manifested by microglial infiltration and astrogliosis linked… Continue reading Alzheimer’s disease (AD) is pathologically seen as a accumulation of β-amyloid